Chronic gastritis with intestinal metaplasia, negative for dysplasia or malignancy.

慢性胃炎伴肠化，无异型增生或恶性肿瘤。

Diffuse intestinal metaplasia has long been considered to be a risk factor for bladder adenocarcinoma.

这种弥漫性肠上皮化生有发生膀胱腺癌的危险.

Gastric cancer usually arises in chronic stomach inflammation and where atrophy and intestinal metaplasia have supervened .

胃癌发生的过程通常是长期的慢性发炎，慢慢导致细胞的萎缩和化生，然后走向癌化。

H pylori chronic active gastritis, gastric atrophy, intestinal metaplasia, and dysplasia are well-recognized sequential events in the development of intestinal-type gastric adenocarcinoma.

在肠型胃腺癌的发展过程中，普遍认为幽门螺杆菌顺序引起胃炎、胃萎缩、肠化生和不典型增生。

It indicated that HP infection may be one of the factors to induce intestinal metaplasia and so to increase the risk of carcinoma of stomach.

提示HP感染是肠上皮化生的促进因素之一，继而增加患胃癌的危险性。

Objective:To investigate the relationship between the development of gastric cancer and different subtypes and local patterns of intestinal metaplasia .

目的：研究肠化的局部型式及不同表型与胃癌发生的关系。

Conclusion Gastroscopy and follow-up of patients with intestinal metaplasia or gastric mucosa dysplasia help to detect gastric carcinoma, especially early-stage gastric carcinoma.

结论对胃黏膜异型增生和肠上皮化生等病变患者的胃镜随访有利于提高胃癌（特别是早期胃癌）的检出率。