Objective: Study the correlation between t cell anergy and apoptosis.

目的：探讨无能T细胞与凋亡的关系。

Clonal anergy and suppressor cells are main mechanisms of the tolerance.

克隆不应答和抑制细胞是耐受的主要机制。

To study the induced condition and characteristics of T cell anergy in vitro.

目的：在体外探讨T细胞无能诱导的条件及其生物学特性。

Aim To explore the molecular mechanism of t cell anergy induced by superantigen SEA.

目的探讨超抗原诱导T细胞无能的分子机制。

Regulatory DCs can induce regulatory T cells, inhibit T cell proliferation or induce T cell anergy.

目前发现， 调节性DC可诱导调节性T细胞产生，抑制T细胞增殖或诱导T细胞增殖阻滞。

The enhanced cell surface expression of CTLA-4 is likely responsible for the induction of t cell anergy.

结论CTLA - 4表面表达水平的升高很可能与T细胞无能状态的诱导有关。

Introduces the concepts of available anergy and reduced exergy by analysing the energy utilization process.

通过对其能量利用过程的分析，引入可用和折合的概念。

Howeer, some T cells are not actiated, in fact they are inactiated by a process called anergy or tolerance.

然而，有些T细胞处于未激发状态，事实上他们被称为无反应性，耐受性的过程灭活了。

Howeer, some T cells are not actiated, in fact they are inactivated by a process called anergy or tolerance.

然而，有些T细胞处于未激发状态，事实上他们被称为无反应性，耐受性的过程灭活了。

However, some t cells are not activated, in fact they are inactivated by a process called anergy or tolerance.

然而，有些T细胞处于未激发状态，事实上他们被称为无反应性，耐受性的过程灭活了。

CD47 induces costimulatory signals on activation of T cell, T cell apoptosis and T cell anergy and enhances the efficiency of TCR signaling;

CD4 7是T细胞活化的共刺激因子，活化T细胞凋亡过程，诱导T细胞无反应性，加强TCR信号传导的效率；

Several other mechanisms may by theorized for the induction of tolerance in this renal transplant model, including clonal anergy and suppressive cells.

结论嵌合状态与肾移植耐受存在明显相关性，克隆不应答、抑制细胞参与了耐受的形成。

The IL2 reverse test and detection of microchimerism showed that the tolerance mechanism involved anergy, and was related to formation of microchimerism.

IL 2逆转实验、微嵌合体检测表明，该耐受与克隆失活、微嵌合体的存在有关。

Given that severe trauma effects as many as 20 percent of the 20,000 or so human genes, finding the genes and proteins involved in t cell anergy and apoptosis amid this storm has been a challenge.

如果大面积损伤影响多达人类基因(大概20000个)中20%的基因的话，找到其中涉及T细胞无能和凋亡的基因和蛋白是一个挑战。