This rodent proof of principle suggests that turning off BCL11A in people with sickle cell disease should allow them to produce enough fetal hemoglobin to reduce or eliminate their symptoms.

小白鼠的实验表明，关闭镰状贫血症患者的BCL11A基因可以让他们体内产生足够的胎儿血红蛋白，从而降低或消灭疾病症状。