Researchers are developing drugs to target amyloid plaques.

研究人员正在开发针对淀粉样蛋白斑块的药物。

PIB adheres to amyloid plaques in the brain.

匹兹堡化合物B结合在脑组织淀粉样斑点上。

It was too late. Think of amyloid plaques as a lit match.

这就为时已晚。试想淀粉样斑块是已经点燃的火柴。

Let's imagine that your amyloid plaques have reached that tipping point.

假设你的淀粉样斑块已经达到临界点。

So what happens when amyloid plaques accumulate to this tipping point?

所以当淀粉样斑块积累到临界点后会发生什么？

Intriguingly, the patients brains were riddled with tangles, but not amyloid plaques.

有趣的是, 患者的大脑中充满了各缠结, 但并没有粉斑.

And when this happens, it binds to itself, forming sticky aggregates called amyloid plaques.

若这一事件发生，突触逐渐束缚了自己，产生一种叫做淀粉样斑块的黏糊糊的聚合体。

Amyloid plaques are one of the characteristic structural abnormalities found in the brains of Alzheimer patients.

淀粉质色斑是在阿兹海默症患者的大脑中所发现的结构异常的特征之一.

This initial step into the disease, this presence of amyloid plaques accumulating, can already be found in your brains.

老年痴呆症的起步，淀粉样斑块的累积，也许已经在你的大脑初步产生。

Definitie diagnosis requires postmortem identification of amyloid plaques and neurofibrillary tangles linked to the disease.

最终确诊需要尸检发现与本病相关的淀粉样蛋白斑及神经纤维缠结.