Objective To explore the specific pathogenic role of glycation products in normal kidney.

目的为探讨糖基化产物对正常肾脏的特异性损害作用。

Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.

结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。

The glycation effect caused by glyoxal, methylglyoxal and other saccharide derivatives is toxic to cells.

糖代谢的衍生物甲基乙二醛等可通过糖化作用产生细胞毒性。

RESULTS Inactivation of esterase was induced by glycation, carbamylation and steroid in a concentration .

结果糖化、氨甲酰化和类固醇诱导酯酶失活的作用呈浓度依赖性，类固醇失活效应最快。

AIM To establish a rapid method for screening advanced glycation end product (AGE) cross links breaker in vitro.

目的建立一种快速、体外筛选晚期糖基化终产物(age)交联结构裂解剂的方法。

An elevated level of skin advanced glycation end products (AGE) is a biomarker of diabetes, the authors explain.

作者解释道，升高的皮肤晚期糖基化终末产物(age)是糖尿病的生物标志物。

CONCLUSION Aspirin and ibuprofen show partial protection against the glycation induced inactivation of SOD and cat.

结论阿斯匹林和布洛芬可部分保护糖基化诱导CAT和SOD的失活。

Hyperglycemia contributes to greater platelet reactivity through direct effects and by promoting glycation of platelet proteins.

高血糖症通过直接影响和促进血小板蛋白的糖化从而增强血小板的活性。

The excess in blood glucose reacts with proteins in tissues to form what are known as advanced glycation end products, or AGE's.

过量血糖反应蛋白在组织中，以何种形式被称为晚期糖化终产物，或年龄的。

Objective to investigate the inhibitory effects of taurine on oxidative stress and nonenzymatic glycation in kidney of diabetic rats.

目的研究牛磺酸对糖尿病大鼠肾脏氧化应激反应和非酶糖基化的抑制效果。

Antibodies that bind specifically to receptor for advanced glycation end products (RAGE) and RAGE-binding fragments thereof are disclosed.

本发明揭示与晚期糖化终产物受体(RAGE)特异性结合的抗体及其RAGE结合片段。

Diabetes is characterized by chronic hyperglycemia which facilitates the irreversible formation of advanced glycation end products (AGEs).

糖尿病以慢性高血糖导致不可逆转的终末糖基化产物(AGEs)形成为特点。

Glycation and other non-enzymic post-translational modifications of proteins have been implicated in the complications of diabetes and other conditions.

糖基化和其他非酶翻译后修饰的蛋白质有牵连在糖尿病并发症和其他条件。

A carb rich diet causes rapid elevation of blood sugars which promotes glycation where sugar molecules bind to a protein such as skin collagen and damage it.

一顿含丰富碳水化合物的大餐，可以让你的血糖水平快速上升，那会促进糖分子和蛋白质，像皮肤必须的胶原所结合，从而毁坏皮肤。

Many studies show that nonenzymatic glycation and glucose autoxidation which are accompanied by generation of free radicals is a process of glycation and oxidation.

许多研究表明非酶糖化和葡萄糖自身氧化伴随着游离基的产生，是一个糖化氧化过程。

Skin autofluorescence can measure tissue for the accumulation of advanced glycation end products (AGEs), which accumulate as a result of high levels of blood sugar.

皮肤自发荧光技术可以测定组织中晚期糖基化终末产物（AGEs）的含量，这正是血糖累积等级的结果。

To construct human mutant CD59(HMCD59) eukaryot ic expression system and investigate whether glycation could inhibit the protection role of HMCD59 against human complement.

构建突变人CD59分子（HMCD59）真核表达体系，探讨HMCD59糖基化前后抗补体活性的变化。

Effects of carbon sources, nitrogen sources and growth factors on the bioactivity inhibition of non-enzymatic glycation reaction of biotransformation products of Ginkgo biloba L.

试验以抑制非酶糖基化反应活性为指标，通过单因素试验筛选了适宜碳源、氮源、无机盐以及复合营养因子，通过正交试验进一步优化培养基组成。

First stage: yeast fermentation: yeast fermentation stage will decompose large molecules into small ones, decompose starch into carbon dioxide and alcohol, also known as glycation.

第一阶段：酵母菌发酵：酵母菌发酵阶段将大分子分解成小分子，把淀粉分解成二氧化碳和酒精，也叫糖化作用。

Objective To study the deleterious effect of glycation end products on normal rat renal structure and function and inhibiting effect of Polyrhachis vicina roger extraction on glycation.

目的研究糖基化终末产物对正常大鼠肾脏功能和结构的有害作用及拟黑多刺蚁醇提物对糖基化的抑制作用。

Once in the bloodstream, sugar bonds with protein and creates advanced glycation end products (aptly abbreviated AGEs), which cause collagen to become inflamed and stiff, leading to wrinkles.

一旦进入血液中，糖分就会与蛋白质融合产生糖基化终产物(缩写为ages)。AGEs导致胶原蛋白挥发变硬产生皱纹。

Results: as compared with positive drug aminoguanidine, the sera of rats treated with TSA showed significantly inhibitory effect on glycation in vitro, and the relative inhibitory rate was 102 2%.

结果：与阳性药氨基胍相比，灌服糖肾安的大鼠血清对体外糖基化反应有明显的抑制作用，相对抑制率为1 0 2 . 2 %。

Objective to investigate the effects of valsartan on the expression of receptors for advanced glycation end-products (RAGE) in kidneys of diabetic rats, and to explore its renoprotection mechanisms.

目的探讨缬沙坦对糖尿病大鼠肾脏糖化终末产物受体(RAGE)表达的影响，探索其肾保护机制。

Objective To investigate the effects of advanced glycation end products (AGE) on secretion of monocyte chemoattractant protein-1(MCP-1) by human endothelial cells and its signal transduction pathway.

目的研究晚期糖基化终产物（AGE）修饰蛋白对人内皮细胞分泌单核细胞趋化蛋白1（MCP 1）的影响及其作用的信号传导途径。